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[DOI] [PMC free article] [PubMed] [Google Scholar] 82.Ikushima I, Jensen L, Flint A, Nishida T, Zacho J, Irie S
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Therapeutic strategies targeting cuproptosis in KOA Copper chelators, such as TTM and D-penicillamine, effectively sequester intracellular excess copper, thereby attenuating copper overload, suppressing FDX1-mediated cuproptosis, and preserving chondrocyte viability
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